Search results for "acute lung injury"

showing 10 items of 28 documents

Novel technologies to detect atelectotrauma in the injured lung

2010

ABSTRACTCyclical recruitment and derecruitment of lung parenchyma (R/D) remains a serious problem in ALI/ARDS patients, defined as atelectotrauma. Detection of cyclical R/D to titrate the optimal respiratory settings is of high clinical importance. Image-based technologies that are capable of detecting changes of lung ventilation within a respiratory cycle include dynamic computed tomography (dCT), synchrotron radiation computed tomography (SRCT), and electrical impedance tomography (EIT). Time-dependent intra-arterial oxygen tension monitoring represents an alternative approach to detect cyclical R/D, as cyclical R/D can result in oscillations of PaO2 within a respiratory cycle. Continuous…

Diagnostic ImagingPulmonary and Respiratory MedicinePathologymedicine.medical_specialtyARDSAcute Lung InjuryClinical BiochemistrySeverity of Illness IndexPredictive Value of TestsImage Interpretation Computer-AssistedElectric ImpedancemedicineHumansOximetryRespiratory systemMolecular BiologyElectrical impedance tomographyRespiratory Distress SyndromeLungmedicine.diagnostic_testbusiness.industryOxygenationrespiratory systemPrognosismedicine.diseaseRespiration Artificialrespiratory tract diseasesOxygen tensionPulse oximetrymedicine.anatomical_structureTomographyTomography X-Ray ComputedbusinessBiomedical engineering
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Cigarette smoke alters IL-33 expression and release in airway epithelial cells

2014

AbstractAirway epithelium is a regulator of innate immune responses to a variety of insults including cigarette smoke. Cigarette smoke alters the expression and the activation of Toll Like Receptor 4 (TLR4), an innate immunity receptor. IL-33, an alarmin, increases innate immunity Th2 responses. The aims of this study were to explore whether mini-bronchoalveolar lavage (mini-BAL) or sera from smokers have altered concentrations of IL-33 and whether cigarette smoke extracts (CSE) alter both intracellular expression (mRNA and protein) and release of IL-33 in bronchial epithelial cells. The role of TLR4 in the expression of IL-33 was also explored.Mini-BALs, but not sera, from smokers show red…

Bronchial epithelial cellLipopolysaccharidesBlotting WesternBronchiInflammationRespiratory MucosaBiologyReal-Time Polymerase Chain ReactionBronchoalveolar LavageImmunoenzyme TechniquesBronchial epithelial cell; COPD; Cigarette smoke; IL-33; InflammationSmokeacute lung injury cigarette smokeinterleukin 33medicineCOPDHumansRNA MessengerReceptorMolecular BiologyCells CulturedCell ProliferationInflammationToll-like receptorInnate immune systemReverse Transcriptase Polymerase Chain ReactionInterleukinsCigarette smokeFlow CytometryInterleukin-33Immunity Innaterespiratory tract diseasesCell biologyToll-Like Receptor 4Interleukin 33ImmunologyIL-33TLR4Molecular MedicineRespiratory epitheliummedicine.symptomIntracellularBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Optimising experimental research in respiratory diseases: an ERS statement

2018

Experimental models are critical for the understanding of lung health and disease and are indispensable for drug development. However, the pathogenetic and clinical relevance of the models is often unclear. Further, the use of animals in biomedical research is controversial from an ethical perspective.The objective of this task force was to issue a statement with research recommendations about lung disease models by facilitating in-depth discussions between respiratory scientists, and to provide an overview of the literature on the available models. Focus was put on their specific benefits and limitations. This will result in more efficient use of resources and greater reduction in the numb…

Animal Experimentation0301 basic medicinePulmonary and Respiratory Medicinemedicine.medical_specialtyBiomedical ResearchStatement (logic)Advisory CommitteeseducationMEDLINEDiseaseLung injury[SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tractHIGHLAND WHITE TERRIERSACUTE LUNG INJURY03 medical and health sciencesIdiopathic pulmonary fibrosis0302 clinical medicineINBRED MOUSE STRAINSPulmonary fibrosisAnimalsHumansMedicineIntensive care medicineSocieties MedicalALLERGIC AIRWAY INFLAMMATIONbusiness.industryCOMMUNITY-ACQUIRED PNEUMONIACIGARETTE-SMOKE EXPOSURERespiration Disordersmedicine.diseaseDISTRESS-SYNDROME3. Good healthEuropeDisease Models Animal030104 developmental biologyANIMAL-MODELS030228 respiratory systemDrug developmentData qualityARTERIAL-HYPERTENSIONIDIOPATHIC PULMONARY-FIBROSISbusiness
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Transmission of arterial oxygen partial pressure oscillations to the cerebral microcirculation in a porcine model of acute lung injury caused by cycl…

2013

Cyclic recruitment and derecruitment (R/D) play a key role in the pathomechanism of acute lung injury (ALI) leading to respiration-dependent oscillations of arterial partial pressure of oxygen (Pa(O(2))). These Pa(O(2)) oscillations could also be forwarded to the cerebral microcirculation.In 12 pigs, partial pressure of oxygen was measured in the thoracic aorta (Pa(O(2))) and subcortical cerebral tissue (Pbr(O(2))). Cerebral cortical haemoglobin oxygen saturation (Sbr(O(2))), cerebral blood flow (CBF), and peripheral haemoglobin saturation (Sp(O(2))) were assessed by spectroscopy and laser Doppler flowmetry. Measurements at different fractions of inspired oxygen (F(I(O(2)))) were performed …

medicine.medical_specialtySwineAcute Lung Injurychemistry.chemical_elementBlood PressureLung injuryAnesthesia GeneralOxygenMicrocirculationInternal medicinemedicineLaser-Doppler FlowmetryAnimalsLungOxygen saturation (medicine)Ventilators Mechanicalbusiness.industryMicrocirculationHemodynamicsLaser Doppler velocimetryRespiration Artificialrespiratory tract diseasesOxygenOxygen Saturation MeasurementAnesthesiology and Pain MedicineBlood pressurechemistryCerebral blood flowAnesthesiaCerebrovascular CirculationCardiologybusinessBlood Gas Monitoring TranscutaneousBronchoalveolar Lavage FluidCraniotomyBritish journal of anaesthesia
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Unexplained chronic liver disease in Ethiopia: a cross-sectional study

2018

Background Hepatitis B virus (HBV) infection is assumed to be the major cause of chronic liver disease (CLD) in sub-Saharan Africa. The contribution of other aetiological causes of CLD is less well documented and hence opportunities to modulate other potential risk factors are being lost. The aims of this study were to explore the aetiological spectrum of CLD in eastern Ethiopia and to identify plausible underlying risk factors for its development. Methods A cross-sectional study was undertaken between April 2015 and April 2016 in two public hospitals in Harar, eastern Ethiopia. The study population comprised of consenting adults with clinical and radiological evidence of chronic liver dise…

MaleCross-sectional studyEpidemiologyBiopsyChronic liver disease0302 clinical medicineRisk FactorsKhatEpidemiologyPrevalenceSIMPLE NONINVASIVE INDEX030212 general & internal medicineViral hepatitisPOPULATIONbiologymedicine.diagnostic_testSub-Saharan AfricaLiver DiseasesGastroenterologyCHRONIC HEPATITISGeneral MedicineAlcoholismLiverLiver biopsyPopulation studyFemale030211 gastroenterology & hepatologyC HEPATITISViral hepatitisLife Sciences & BiomedicineResearch ArticleAdultmedicine.medical_specialtySubstance-Related DisordersAcute Lung InjuryKHAT LEAVESAUTOIMMUNEVERBAL AUTOPSY METHODCathaVIRUS-INFECTIONCatha edulis03 medical and health sciencesInternal medicinemedicineHumanslcsh:RC799-869Science & TechnologyGastroenterology & HepatologySIGNIFICANT FIBROSISbusiness.industryHepatotoxicity1103 Clinical SciencesHepatologybiology.organism_classificationmedicine.diseaseCross-Sectional StudiesChronic Diseaselcsh:Diseases of the digestive system. GastroenterologyEthiopiabusiness
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Pre-treatment with mesenchymal stem cells reduces ventilator-induced lung injury

2012

ckground information: Bone marrow-derived mesenchymal stem cells (MSCs) reduce acute lung injury in animals challenged by bleomycin or bacterial lipopolysaccaride. It is not known, however, whether MSCs protect from ventilator- induced lung injury (VILI). Question of the study: Whether MSCs have a potential role in preventing or modulating VILI in healthy rats subjected to high-volume ventilation. Materials and methods: 24 Sprague-Dawley rats (250-300 g) were subjected to high- volume mechanical ventilation (25 ml/kg). MSCs (5x106) were intravenously or intratracheally administered (N=8 each) 30 min before starting over-ventilation and 8 rats were MSC-untreated. Spontaneously breathing anes…

MalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyAdministration TopicalVentilator-Induced Lung Injurymedicine.medical_treatmentVascular Cell Adhesion Molecule-1Pulmonary EdemaSettore MED/10 - Malattie Dell'Apparato RespiratorioLung injuryMesenchymal Stem Cell TransplantationBleomycinRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAcute lung injury cell therapy injurious ventilation lung edema lung inflammation mechanical ventilationAnimalsMedicineMacrophageCell adhesionLung030304 developmental biologyMechanical ventilation0303 health sciencesmedicine.diagnostic_testbusiness.industryMesenchymal stem cellrespiratory systemRatsrespiratory tract diseasesDisease Models AnimalBronchoalveolar lavage030228 respiratory systemchemistryBreathingAdministration IntravenousInflammation MediatorsbusinessBronchoalveolar Lavage FluidEuropean Respiratory Journal
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Targeted-lung delivery of dexamethasone using gated mesoporous silica nanoparticles. A new therapeutic approach for acute lung injury treatment

2021

Acute lung injury (ALI) is a critical inflammatory syndrome, characterized by increased diffuse inflammation and severe lung damage, which represents a clinical concern due to the high morbidity and mortality in critical patients. In last years, there has been a need to develop more effective treatments for ALI, and targeted drug delivery to inflamed lungs has become an attractive research field. Here, we present a nanodevice based on mesoporous silica nanoparticles loaded with dexamethasone (a glucocorticoid extensively used for ALI treatment) and capped with a peptide that targets the TNFR1 receptor expressed in pro-inflammatory macrophages (TNFR-Dex-MSNs) and avoids cargo leakage. TNFR-D…

Acute Lung InjuryPharmaceutical ScienceInflammationLung injuryPharmacologyDexamethasoneMiceIn vivomedicineAnimalsHumansLungDexamethasoneLungbusiness.industryrespiratory systemSilicon Dioxiderespiratory tract diseasesmedicine.anatomical_structureTargeted drug deliveryNanoparticlesTumor necrosis factor alphamedicine.symptombusinessGlucocorticoidmedicine.drugJournal of Controlled Release
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TLR4 Up-regulation and Reduced Foxp3 Expression in Mechanically Ventilated Smokers with Obstructive Chronic Bronchitis

2013

Background: Chronic bronchitis (CB) is a risk factor in chronic obstructive pulmonary disease (COPD) for accelerated lung function decline and increased mortality. The lung and systemic inflammatory and immunological profile of COPD patients with CB which acutely experience respiratory failure upon a disease exacerbation is unknown. Methods: In this study, we explored the expression of Foxp3 by western blot analysis, TLR4 by immunocytochemistry and the concentrations of IP-10 and IL-8 by ELISA in the mini-bronchoalveolar lavages (mini-BAL) and in the peripheral blood of patients with respiratory failure requiring intubation and mechanical ventilation. The recruited subjects were separated i…

Pulmonary and Respiratory MedicineMaleChronic bronchitismedicine.medical_specialtyPathologyNeutrophilsmedicine.medical_treatmentSettore MED/41 - AnestesiologiaGastroenterologyStatistics NonparametricLeukocyte CountPulmonary Disease Chronic ObstructiveAcute Lung InjuryToll-like receptors Foxp3 Chemokines Smokers Respiratory failureInternal medicineMedicineHumansRisk factorAgedMechanical ventilationAged 80 and overCOPDLungbusiness.industryInterleukin-8SmokingFOXP3Forkhead Transcription Factorsmedicine.diseaseRespiration Artificialrespiratory tract diseasesUp-RegulationBronchitis ChronicChemokine CXCL10Toll-Like Receptor 4medicine.anatomical_structureRespiratory failureBronchitisFemalebusinessBronchoalveolar Lavage Fluid
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CD11c+ Alveolar Macrophages are a Source of IL-23 During Lipopolysaccharide-Induced Acute Lung Injury

2013

Acute lung injury (ALI) is a severe pulmonary disease causing high numbers of fatalities worldwide. Innate immune responses are an integral part of the pathophysiologic events during ALI. Interleukin 23 (IL-23) is a proinflammatory mediator known to direct the inflammatory responses in various settings of infection, autoimmunity, and cancer. Interleukin 23 has been associated with proliferation and effector functions in T(H)17 cells. Surprisingly, little is known about production of IL-23 during ALI. In this study, we found expression of mRNA for IL-23p19 to be 10-fold elevated in lung homogenates of C57BL/6 mice after lipopolysaccharide (LPS)-induced ALI. Likewise, concentrations of IL-23 …

LipopolysaccharidesMaleLipopolysaccharideAcute Lung InjuryCD11cBiologyLung injuryCritical Care and Intensive Care MedicineInterleukin-23ArticleProinflammatory cytokineMicechemistry.chemical_compoundMacrophages AlveolarmedicineAnimalsInnate immune systemmedicine.diagnostic_testrespiratory systemCD11c Antigenrespiratory tract diseasesBronchoalveolar lavagechemistryImmunologyEmergency MedicineAlveolar macrophageInterleukin 17Shock
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Transfusion-related acute lung injury after intravenous immunoglobulin treatment in a lung transplant recipient

2012

Three weeks after single-lung transplantation for pulmonary fibrosis, a patient with high serum levels of de novo donor-specific antibodies received high-dose intravenous immunoglobulin (IVIG) infusion (scheduled dose: 2 g/kg on 2 days) to prevent antibody-mediated rejection. Within the first hours after completion of infusions, he experienced acute lung injury involving the transplanted lung. Given the clinical evolution and the absence of an alternative diagnosis, transfusion-related acute lung injury (TRALI) was diagnosed. The IVIG administered on each day was from the same batch. At day 110, because of an increase in the serum titers of donor-specific antibodies, IVIG therapy was reintr…

medicine.medical_specialtybiologybusiness.industrymedicine.medical_treatmentHematologyGeneral MedicineLung injurymedicine.diseaseSurgeryTransplantationhemic and lymphatic diseasesAnesthesiaPulmonary fibrosismedicinebiology.proteinLung transplantationLung transplant recipientAntibodybusinessIVIG TherapyTransfusion-related acute lung injuryVox Sanguinis
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